The article presents a computer simulation model for adaptive bone remodeling based on the adaptive bone-remodeling theory and the Finite Element Method (FEM). The model uses strain energy density (SED) as a feedback control variable to predict bone remodeling in response to stress changes. The study investigates the relationship between 'stress shielding' and bone resorption around intramedullary prostheses, such as femoral stems in Total Hip Arthroplasty (THA). It shows that bone resorption depends mainly on the rigidity and bonding characteristics of the implant. The model is applied to investigate the effects of implant rigidity and bonding characteristics on bone remodeling. The results indicate that the most flexible stem causes bone resorption at the proximal end, similar to 'calcar resorption' in clinical orthopaedics. The study also shows that the resorption process is more effective in external remodeling than in internal remodeling. The results are compatible with animal experimental data on similar intramedullary configurations. The study concludes that 'stress shielding' is a transient phenomenon, and that a homeostatic strain configuration can be achieved by the bone if the stem is not too rigid and the bone resorption process occurs mainly at the periosteal surface. The study provides quantitative guidelines for determining whether a stem is too rigid to make bone-strain normalisation likely.The article presents a computer simulation model for adaptive bone remodeling based on the adaptive bone-remodeling theory and the Finite Element Method (FEM). The model uses strain energy density (SED) as a feedback control variable to predict bone remodeling in response to stress changes. The study investigates the relationship between 'stress shielding' and bone resorption around intramedullary prostheses, such as femoral stems in Total Hip Arthroplasty (THA). It shows that bone resorption depends mainly on the rigidity and bonding characteristics of the implant. The model is applied to investigate the effects of implant rigidity and bonding characteristics on bone remodeling. The results indicate that the most flexible stem causes bone resorption at the proximal end, similar to 'calcar resorption' in clinical orthopaedics. The study also shows that the resorption process is more effective in external remodeling than in internal remodeling. The results are compatible with animal experimental data on similar intramedullary configurations. The study concludes that 'stress shielding' is a transient phenomenon, and that a homeostatic strain configuration can be achieved by the bone if the stem is not too rigid and the bone resorption process occurs mainly at the periosteal surface. The study provides quantitative guidelines for determining whether a stem is too rigid to make bone-strain normalisation likely.