The article discusses the influence of TOR kinase on lifespan in *Caenorhabditis elegans*. The absence of LET-363/TOR activity causes developmental arrest at the L3 larval stage, but when TOR is inhibited using RNA interference, the worms exhibit a significantly extended mean lifespan. This extension is comparable to that observed in *C. elegans* mutants with reduced DAF-2/IGF signaling, suggesting a potential link between TOR and the DAF-2/IGF pathway in controlling ageing, metabolism, and reproductive growth. The findings also indicate that TOR may act downstream or independently of the forkhead transcription factor DAF-16, and that it interacts with the insulin endocrine system. The study provides insights into the regulation of lifespan by nutrient sensing and insulin signaling in nematodes.The article discusses the influence of TOR kinase on lifespan in *Caenorhabditis elegans*. The absence of LET-363/TOR activity causes developmental arrest at the L3 larval stage, but when TOR is inhibited using RNA interference, the worms exhibit a significantly extended mean lifespan. This extension is comparable to that observed in *C. elegans* mutants with reduced DAF-2/IGF signaling, suggesting a potential link between TOR and the DAF-2/IGF pathway in controlling ageing, metabolism, and reproductive growth. The findings also indicate that TOR may act downstream or independently of the forkhead transcription factor DAF-16, and that it interacts with the insulin endocrine system. The study provides insights into the regulation of lifespan by nutrient sensing and insulin signaling in nematodes.