This meta-analysis of 41 functional neuroimaging studies examines the neural correlates of executive function in schizophrenia. The study aims to determine whether common nodes of the cognitive control network exhibit altered activity and to evaluate the direction of these effects. The analysis included studies that used functional neuroimaging during executive function tasks in adult patients with schizophrenia and healthy controls. The results show that both healthy controls and patients activate a similar cortical-subcortical network, including the dorsolateral prefrontal cortex (DLPFC), ventrolateral PFC, anterior cingulate cortex (ACC), and thalamus. However, patients with schizophrenia show reduced activation in the left DLPFC, rostral/dorsal ACC, left thalamus, and inferior/posterior cortical areas. Increased activation was observed in several midline cortical areas. The findings suggest that healthy adults and schizophrenic patients activate a qualitatively similar neural network during executive tasks, consistent with the engagement of a general-purpose cognitive control network. However, patients with schizophrenia exhibit altered activity, particularly in the DLPFC, ACC, and mediodorsal thalamus, indicating deficits in this network. These results have implications for understanding the pathophysiology of cognitive pathology in schizophrenia and for developing targeted therapies.This meta-analysis of 41 functional neuroimaging studies examines the neural correlates of executive function in schizophrenia. The study aims to determine whether common nodes of the cognitive control network exhibit altered activity and to evaluate the direction of these effects. The analysis included studies that used functional neuroimaging during executive function tasks in adult patients with schizophrenia and healthy controls. The results show that both healthy controls and patients activate a similar cortical-subcortical network, including the dorsolateral prefrontal cortex (DLPFC), ventrolateral PFC, anterior cingulate cortex (ACC), and thalamus. However, patients with schizophrenia show reduced activation in the left DLPFC, rostral/dorsal ACC, left thalamus, and inferior/posterior cortical areas. Increased activation was observed in several midline cortical areas. The findings suggest that healthy adults and schizophrenic patients activate a qualitatively similar neural network during executive tasks, consistent with the engagement of a general-purpose cognitive control network. However, patients with schizophrenia exhibit altered activity, particularly in the DLPFC, ACC, and mediodorsal thalamus, indicating deficits in this network. These results have implications for understanding the pathophysiology of cognitive pathology in schizophrenia and for developing targeted therapies.