This short report describes eight patients who presented with segmental necrotizing glomerulonephritis and a generalized illness over a five-year period. The clinical findings and geographic distribution suggest that infection by a group A arbovirus, particularly Ross River virus, may have been a contributing factor. All patients had microscopic hematuria, proteinuria, and raised urine sediment counts. Renal biopsies revealed segmental necrotizing glomerulonephritis with fibrin accumulation and crescent formation. Treatment with prednisolone and cyclophosphamide or azathioprine was effective, and most patients recovered fully, although some experienced recurrent disease. The authors suspect that the glomerular lesions were related to arbovirus hepatitis, possibly caused by Ross River virus, based on serological evidence of previous infection in seven out of eight patients. Further serological investigation is needed to confirm the causal role of Ross River virus in this form of glomerulonephritis.This short report describes eight patients who presented with segmental necrotizing glomerulonephritis and a generalized illness over a five-year period. The clinical findings and geographic distribution suggest that infection by a group A arbovirus, particularly Ross River virus, may have been a contributing factor. All patients had microscopic hematuria, proteinuria, and raised urine sediment counts. Renal biopsies revealed segmental necrotizing glomerulonephritis with fibrin accumulation and crescent formation. Treatment with prednisolone and cyclophosphamide or azathioprine was effective, and most patients recovered fully, although some experienced recurrent disease. The authors suspect that the glomerular lesions were related to arbovirus hepatitis, possibly caused by Ross River virus, based on serological evidence of previous infection in seven out of eight patients. Further serological investigation is needed to confirm the causal role of Ross River virus in this form of glomerulonephritis.