YTHDF3 facilitates translation and decay of N6-methyladenosine-modified RNA

YTHDF3 facilitates translation and decay of N6-methyladenosine-modified RNA

20 January 2017 | Hailing Shi1,2,* , Xiao Wang1,2,* , Zhike Lu1,2, Boxuan S Zhao1,2, Honghui Ma1,2, Phillip J Hsu1,2,3, Chang Liu1,2, Chuan He1,2,4
The study investigates the role of YTHDF3, a cytoplasmic m6A-binding protein, in the regulation of mRNA metabolism. YTHDF3 is shown to facilitate protein synthesis in synergy with YTHDF1 and affect mRNA decay mediated by YTHDF2. YTHDF3 binds m6A-modified mRNAs and shares common targets with YTHDF1 and YTHDF2. Depletion of YTHDF3 reduces translation efficiency of its targets and affects the RNA-binding specificity of YTHDF1 and YTHDF2. YTHDF3 functions as a hub for partitioning m6A-modified transcripts among the three proteins, potentially affecting mRNA stability and translation. The results suggest that YTHDF3 plays a critical role in coordinating the metabolism of m6A-modified mRNAs in the cytoplasm.The study investigates the role of YTHDF3, a cytoplasmic m6A-binding protein, in the regulation of mRNA metabolism. YTHDF3 is shown to facilitate protein synthesis in synergy with YTHDF1 and affect mRNA decay mediated by YTHDF2. YTHDF3 binds m6A-modified mRNAs and shares common targets with YTHDF1 and YTHDF2. Depletion of YTHDF3 reduces translation efficiency of its targets and affects the RNA-binding specificity of YTHDF1 and YTHDF2. YTHDF3 functions as a hub for partitioning m6A-modified transcripts among the three proteins, potentially affecting mRNA stability and translation. The results suggest that YTHDF3 plays a critical role in coordinating the metabolism of m6A-modified mRNAs in the cytoplasm.
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