miR396b/GRF6 module contributes to salt tolerance in rice

miR396b/GRF6 module contributes to salt tolerance in rice

2024 | Huanran Yuan1,2,†, Mingxing Cheng1,2,†, Ruihua Wang1, Zhikai Wang3, Fengfeng Fan1,2, Wei Wang1, Fengfeng Si1, Feng Gao1 and Shaoqing Li1,2,*
Salinity is a significant environmental challenge that threatens global food security. This study investigates the role of the miR396b/GRF6 module in enhancing salt tolerance in rice. The authors found that the miR396b/GRF6 module significantly improved salt tolerance in rice, with transgenic lines showing a 48.0% and 74.4% increase in survival rates compared to the wild type. The module also reduced H2O2 accumulation and increased the activities of ROS-scavenging enzymes (CAT, SOD, and POD). The study identified ZNF9, a negative regulator of rice salt tolerance, which directly binds to the promoter of miR396b to modulate its expression. Transcriptome and ChIP-seq analysis revealed that MYB3R serves as a downstream target of miR396b/GRF6 and its overexpression significantly enhanced salt tolerance. These findings provide valuable insights into the molecular mechanisms underlying the response of the miR396b/GRF6 network to salt stress in rice, offering potential genetic resources for breeding high-yield, salt-tolerant rice varieties.Salinity is a significant environmental challenge that threatens global food security. This study investigates the role of the miR396b/GRF6 module in enhancing salt tolerance in rice. The authors found that the miR396b/GRF6 module significantly improved salt tolerance in rice, with transgenic lines showing a 48.0% and 74.4% increase in survival rates compared to the wild type. The module also reduced H2O2 accumulation and increased the activities of ROS-scavenging enzymes (CAT, SOD, and POD). The study identified ZNF9, a negative regulator of rice salt tolerance, which directly binds to the promoter of miR396b to modulate its expression. Transcriptome and ChIP-seq analysis revealed that MYB3R serves as a downstream target of miR396b/GRF6 and its overexpression significantly enhanced salt tolerance. These findings provide valuable insights into the molecular mechanisms underlying the response of the miR396b/GRF6 network to salt stress in rice, offering potential genetic resources for breeding high-yield, salt-tolerant rice varieties.
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[slides] miR396b%2FGRF6 module contributes to salt tolerance in rice | StudySpace